Swine diets consisting mainly of corn are at the highest risk of mycotoxin exposure from the decay in the fields or the storage of the corn.
Mycotoxins thrive in the presence of mold on the corn itself, so the areas of the country that have higher moisture have increased risk of mycotoxin introduction.
Mycotoxins are toxic secondary metabolites produced by fungi growing on crops in the field, during handling and in storage.
Molds produce mycotoxins under a wide range of conditions.
They enter animal production system through feed (concentrate, silage or forage) or through bedding.
Some toxins tend to be more common in temperate or tropic climates, but most fungi and mycotoxins are found worldwide.
Mycotoxins unconstructively affect animal performance, animal health and product quality.
Mycotoxin control is critical for production economics and product quality.
Mycotoxins show their effects four different ways: 1.
Intake reduction or feed refusal 2.
Alteration in nutrient content of feed in terms of nutrient absorption and metabolism 3.
Effects on the endocrine and exocrine systems 4.
Suppression of the immune system Mycotoxins display a range of biological outcomes: 1.
Liver and kidney toxicity 2.
Malfunction of the central nervous system 3.
Fertility and reproduction problems 4.
Effect on immunity 5.
Mycotoxins are also deposited in the food supply chain (e.
g.
milk, eggs, meat) and therefore a risk for human health.
Long range goals should be to decrease the amounts of mycotoxin exposure by using mold inhibitors and checking your sources of feed.
It is important to remember that mold inhibitors cannot destroy mycotoxins.
Mold inhibitors are used as feed additives to reduce the level of mold growth in the grain storage facilities, thus minimizing the risk of mycotoxin-producing grains spreading through the feed.
The most commonly used are propionic acid and other organic acids.
However, mold growth prevention does not actually address existing mycotoxins present in contaminated feed.
Mycotoxin binders are substances that actually capture the mycotoxins in the system from the mold grain exposure and prevent them from being absorbed in the gut and the blood circulation of the animal.
If other attempts at mold containment have failed, then using a mycotoxin binder will take care of the problem.
If it becomes too costly to routinely check for mycotoxins or mold exposure, then regularly adding a mycotoxin binding product will maintain that the livestock will be free of mycotoxicosis.
T-BINDTM has been proven to capture more than just aflatoxin like most other competing products and will stand against other mycotoxins like vomitoxin.
Mycotoxins thrive in the presence of mold on the corn itself, so the areas of the country that have higher moisture have increased risk of mycotoxin introduction.
Mycotoxins are toxic secondary metabolites produced by fungi growing on crops in the field, during handling and in storage.
Molds produce mycotoxins under a wide range of conditions.
They enter animal production system through feed (concentrate, silage or forage) or through bedding.
Some toxins tend to be more common in temperate or tropic climates, but most fungi and mycotoxins are found worldwide.
Mycotoxins unconstructively affect animal performance, animal health and product quality.
Mycotoxin control is critical for production economics and product quality.
Mycotoxins show their effects four different ways: 1.
Intake reduction or feed refusal 2.
Alteration in nutrient content of feed in terms of nutrient absorption and metabolism 3.
Effects on the endocrine and exocrine systems 4.
Suppression of the immune system Mycotoxins display a range of biological outcomes: 1.
Liver and kidney toxicity 2.
Malfunction of the central nervous system 3.
Fertility and reproduction problems 4.
Effect on immunity 5.
Mycotoxins are also deposited in the food supply chain (e.
g.
milk, eggs, meat) and therefore a risk for human health.
Long range goals should be to decrease the amounts of mycotoxin exposure by using mold inhibitors and checking your sources of feed.
It is important to remember that mold inhibitors cannot destroy mycotoxins.
Mold inhibitors are used as feed additives to reduce the level of mold growth in the grain storage facilities, thus minimizing the risk of mycotoxin-producing grains spreading through the feed.
The most commonly used are propionic acid and other organic acids.
However, mold growth prevention does not actually address existing mycotoxins present in contaminated feed.
Mycotoxin binders are substances that actually capture the mycotoxins in the system from the mold grain exposure and prevent them from being absorbed in the gut and the blood circulation of the animal.
If other attempts at mold containment have failed, then using a mycotoxin binder will take care of the problem.
If it becomes too costly to routinely check for mycotoxins or mold exposure, then regularly adding a mycotoxin binding product will maintain that the livestock will be free of mycotoxicosis.
T-BINDTM has been proven to capture more than just aflatoxin like most other competing products and will stand against other mycotoxins like vomitoxin.
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